glutathione peroxidase 1 mechanism peroxidase-1 and neuromodulation: Novel potentials of an old enzyme The role of di-(2-ethylhexyl)

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T2DM develops due to insulin’s inhibitory effect on liver glucose production or due to different defects in insulin secretion

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Notably, breast cancer cells achieve metabolic adaptation by hijacking the autophagy mechanism: under glucose-deprivation conditions, MANF-mediated mitophagy eliminates dysfunctional mitochondria through the PRKN/Parkin-dependent pathway, maintains the ATP supply and promotes tumour survival [112], suggesting that targeting the MANF‒PRKN interaction can break the drug resistance barrier of breast cancer under metabolic stress