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This dysbiotic transformation precipitates compromised SCFA biosynthetic capacity, disruption of intestinal epithelial barrier integrity, and the pathological phenomenon of intestinal hyperpermeability or “leaky gut syndrome.” The consequent translocation of gut-derived pathogen-associated molecular patterns (PAMPs), exemplified by lipopolysaccharides, orchestrates chronic low-grade inflammation through Toll-like receptor (TLR) activation and cascading pro-inflammatory cytokine release, thereby accelerating epithelial-mesenchymal transition within renal tubular epithelium and promoting progressive interstitial fibrogenesis (Rukavina Mikusic et al., 2020

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[DOI] [PMC free article] [PubMed] [Google Scholar] 193.Velazquez R., Ash J.A., Powers B.E., Kelley C.M., Strawderman M., Luscher Z.I., Ginsberg S.D., Mufson E.J., Strupp B.J