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Key Points The biochemical abnormalities that constitute the molecular prodrome of Parkinson disease (PD) can be initiated by genetic or environmental factors and seem to be common to familial and sporadic disease The molecular prodrome will determine the pathology that evolves in PD and the associated cell dysfunction and death The clinical prodrome will, in turn, reflect the evolving molecular and pathological prodromes and offers an important opportunity for early detection and therapeutic intervention Future symptomatic and disease-modifying or neuroprotective drugs are likely to be based on interventions designed to interrupt the evolution of the molecular and pathological prodromes This is a preview of subscription content, access via your institution Access options Subscribe to this journal Receive 12 print issues and online access $189.00 per year only $15.75 per issue Buy this article Purchase on SpringerLink Instant access to the full article PDF

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Ferroptosis is a regulated cell death triggered by oxidative stress that has been