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The glyoxalase system provides an alternative source of mGSH by allowing the intermediate S-d-lactylglutathione to enter the mitochondria and be hydrolyzed by the mitochondrial enzyme glyoxalase II into D-lactate and GSH [73]

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Despite advancements in multimodal treatment, the typical lifespan of GBM patients has only increased by 12–14 months, indicating a minimal enhancement of under 5% [6]