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244 In diabetic complications research, the activation of p53/SLC7A11/GSH axis causes endothelial cell ferroptosis under hyperglycemia, suggesting the important role of ferroptosis of endothelial dysfunction caused by diabetes
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For example, a short initial Th2 response is beneficial to worm expulsion, while a prolonged activation of the type 2 immune response may lead to a shift in the T-cell pool, thereby increasing the number of Treg cells (262)
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