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In a mouse model, the core SWI/SNF subunit BAF155 (SMARCC1) was found to be dispensable for development and growth of skeletal muscle, but loss of BAF155 resulted in enhanced endurance exercise capacity [46]
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This pH change destabilizes the PEG shell on nanoparticles and accelerates drug release (e.g., amikacin) at the site of infection
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